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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Allium sativum var. ophioscorodon
Variétés d'ail à col dur présentant une teneur en allicin plus élevée et des profils aromatiques plus complexes que l'ail à col souple.
Allium sativum var. ophioscorodon, commonly known as hardneck garlic, is a botanical variety characterized by higher allicin content and a more complex flavor profile than softneck garlic. It is traditionally used for cardiovascular support, immune enhancement, and antimicrobial therapy. Key active compounds include allicin, alliin, ajoene, and quercetin, which contribute to its pharmacological effects.
Allicin, the primary sulfur compound, inhibits HMG-CoA reductase, reducing cholesterol synthesis, and exerts antiplatelet effects by blocking thromboxane A2 synthesis and platelet aggregation via ajoene. Allicin also disrupts microbial cell membranes through thiol reactivity, while quercetin provides antioxidant and anti-inflammatory actions by scavenging free radicals and modulating NF-κB pathways. These mechanisms collectively support cardiovascular health, immune function, and antimicrobial activity.
Variétés d'ail à col dur présentant une teneur en allicin plus élevée et des profils aromatiques plus complexes que l'ail à col souple.
Allium sativum var. ophioscorodon, commonly known as hardneck garlic, is a botanical variety characterized by higher allicin content and a more complex flavor profile than softneck garlic. It is traditionally used for cardiovascular support, immune enhancement, and antimicrobial therapy. Key active compounds include allicin, alliin, ajoene, and quercetin, which contribute to its pharmacological effects.
Allicin, the primary sulfur compound, inhibits HMG-CoA reductase, reducing cholesterol synthesis, and exerts antiplatelet effects by blocking thromboxane A2 synthesis and platelet aggregation via ajoene. Allicin also disrupts microbial cell membranes through thiol reactivity, while quercetin provides antioxidant and anti-inflammatory actions by scavenging free radicals and modulating NF-κB pathways. These mechanisms collectively support cardiovascular health, immune function, and antimicrobial activity.