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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Alnus serrulata
Un bouleau d'Amérique du Nord oriental utilisé par les peuples autochtones pour la fièvre, la douleur et comme astringent.
Alnus serrulata, a North American alder, has traditional use among Native Americans for fever, pain, and as an astringent. Modern research indicates anti-inflammatory and wound-healing properties, attributed to its tannin and flavonoid content. Evidence remains limited (Level C), supporting its use in topical and oral preparations.
Tannins in Alnus serrulata exert astringent effects by precipitating proteins on mucosal surfaces, reducing inflammation and promoting wound healing. Flavonoids, such as quercetin, inhibit cyclooxygenase (COX) and lipoxygenase (LOX) pathways, decreasing prostaglandin and leukotriene synthesis, which contributes to anti-inflammatory and analgesic actions. Additionally, these compounds may modulate NF-κB signaling, further attenuating inflammatory cytokine production.
Un bouleau d'Amérique du Nord oriental utilisé par les peuples autochtones pour la fièvre, la douleur et comme astringent.
Alnus serrulata, a North American alder, has traditional use among Native Americans for fever, pain, and as an astringent. Modern research indicates anti-inflammatory and wound-healing properties, attributed to its tannin and flavonoid content. Evidence remains limited (Level C), supporting its use in topical and oral preparations.
Tannins in Alnus serrulata exert astringent effects by precipitating proteins on mucosal surfaces, reducing inflammation and promoting wound healing. Flavonoids, such as quercetin, inhibit cyclooxygenase (COX) and lipoxygenase (LOX) pathways, decreasing prostaglandin and leukotriene synthesis, which contributes to anti-inflammatory and analgesic actions. Additionally, these compounds may modulate NF-κB signaling, further attenuating inflammatory cytokine production.