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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Salvia azurea
Sauge des prairies nord-américaine utilisée par les Amérindiens des Plaines à des fins respiratoires et cérémonielles.
Azure Sage (Salvia azurea) is a North American prairie sage traditionally used by Plains Native Americans for respiratory conditions and ceremonial purposes. Modern research indicates potential antimicrobial and anti-inflammatory activities, attributed to compounds such as flavonoids and terpenoids including rosmarinic acid, cineole, and ursolic acid. It is primarily used as a tea for respiratory support.
The antimicrobial action is largely due to cineole and camphor disrupting bacterial cell membranes, while rosmarinic acid and flavonoids inhibit pro-inflammatory cytokines via NF-κB pathway modulation. Ursolic acid may further contribute by inhibiting COX-2 and LOX enzymes, reducing prostaglandin and leukotriene synthesis. The expectorant effect is mediated by cineole stimulating bronchial secretion and ciliary motility.
Sauge des prairies nord-américaine utilisée par les Amérindiens des Plaines à des fins respiratoires et cérémonielles.
Azure Sage (Salvia azurea) is a North American prairie sage traditionally used by Plains Native Americans for respiratory conditions and ceremonial purposes. Modern research indicates potential antimicrobial and anti-inflammatory activities, attributed to compounds such as flavonoids and terpenoids including rosmarinic acid, cineole, and ursolic acid. It is primarily used as a tea for respiratory support.
The antimicrobial action is largely due to cineole and camphor disrupting bacterial cell membranes, while rosmarinic acid and flavonoids inhibit pro-inflammatory cytokines via NF-κB pathway modulation. Ursolic acid may further contribute by inhibiting COX-2 and LOX enzymes, reducing prostaglandin and leukotriene synthesis. The expectorant effect is mediated by cineole stimulating bronchial secretion and ciliary motility.