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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Broussonetia papyrifera
Arbre d'Asie orientale utilisé en MTC pour les affections du foie et des reins et comme tonique nutritif.
Broussonetia papyrifera, known as paper mulberry, is a traditional East Asian herb used in TCM primarily as a liver and kidney tonic and general nutritive supplement. Modern research suggests hepatoprotective, anti-inflammatory, and antioxidant activities, attributed to compounds such as broussinin, flavonoids, and sterols. It is also employed for its diuretic effects and as a mild adaptogen.
The hepatoprotective effects of B. papyrifera are mediated through activation of the Nrf2/ARE pathway, enhancing antioxidant enzyme expression (e.g., SOD, catalase) and reducing oxidative stress. Its anti-inflammatory activity involves inhibition of NF-κB signaling and downregulation of COX-2 and iNOS expression. The diuretic action may be due to increased renal blood flow and electrolyte excretion, possibly via flavonoid-induced modulation of aquaporins and ion channels.
Arbre d'Asie orientale utilisé en MTC pour les affections du foie et des reins et comme tonique nutritif.
Broussonetia papyrifera, known as paper mulberry, is a traditional East Asian herb used in TCM primarily as a liver and kidney tonic and general nutritive supplement. Modern research suggests hepatoprotective, anti-inflammatory, and antioxidant activities, attributed to compounds such as broussinin, flavonoids, and sterols. It is also employed for its diuretic effects and as a mild adaptogen.
The hepatoprotective effects of B. papyrifera are mediated through activation of the Nrf2/ARE pathway, enhancing antioxidant enzyme expression (e.g., SOD, catalase) and reducing oxidative stress. Its anti-inflammatory activity involves inhibition of NF-κB signaling and downregulation of COX-2 and iNOS expression. The diuretic action may be due to increased renal blood flow and electrolyte excretion, possibly via flavonoid-induced modulation of aquaporins and ion channels.