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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Bryonia alba
Plante grimpante européenne aux racines hautement toxiques, utilisée en homéopathie et prudemment en phytothérapie pour la douleur articulaire.
Bryonia alba is a European climbing plant with highly toxic roots, used primarily in homeopathic preparations for joint pain and pleurisy. Its key active compounds include cucurbitacins, bryonin, and bryonidin, which exhibit anti-inflammatory and analgesic properties, but the raw plant is dangerously toxic and not used in conventional herbal medicine.
Cucurbitacins in Bryonia alba inhibit nuclear factor kappa B (NF-κB) and cyclooxygenase-2 (COX-2), reducing pro-inflammatory cytokine production and prostaglandin synthesis. Additionally, bryonin and bryonidin may modulate leukotriene pathways and suppress neutrophil activation, contributing to anti-inflammatory effects. However, these mechanisms are accompanied by potent gastrointestinal toxicity due to cucurbitacin-induced membrane disruption and apoptosis in enterocytes.
Plante grimpante européenne aux racines hautement toxiques, utilisée en homéopathie et prudemment en phytothérapie pour la douleur articulaire.
Bryonia alba is a European climbing plant with highly toxic roots, used primarily in homeopathic preparations for joint pain and pleurisy. Its key active compounds include cucurbitacins, bryonin, and bryonidin, which exhibit anti-inflammatory and analgesic properties, but the raw plant is dangerously toxic and not used in conventional herbal medicine.
Cucurbitacins in Bryonia alba inhibit nuclear factor kappa B (NF-κB) and cyclooxygenase-2 (COX-2), reducing pro-inflammatory cytokine production and prostaglandin synthesis. Additionally, bryonin and bryonidin may modulate leukotriene pathways and suppress neutrophil activation, contributing to anti-inflammatory effects. However, these mechanisms are accompanied by potent gastrointestinal toxicity due to cucurbitacin-induced membrane disruption and apoptosis in enterocytes.