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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Prunus bokhariensis
Prunes séchées d'Asie centrale utilisées dans la médecine Unani comme laxatifs, pour la fièvre et comme toniques hépatiques.
Bukhara Plum (Prunus bokhariensis) is a dried fruit from Central Asia traditionally used in Unani medicine as a laxative, antipyretic, and liver tonic. Its primary active compounds include sorbitol, malic acid, phenolic acids, and flavonoids, which contribute to its mild laxative and hepatoprotective effects. Evidence is limited to preliminary studies and traditional use, placing it at an evidence level C.
The laxative effect is primarily due to sorbitol, an osmotic agent that draws water into the intestinal lumen, stimulating peristalsis. Phenolic acids and flavonoids may exert hepatoprotective effects by scavenging free radicals and modulating phase II detoxification enzymes, such as glutathione S-transferase. Antipyretic activity is hypothesized to involve inhibition of prostaglandin synthesis via cyclooxygenase (COX) pathways, though specific receptor interactions remain uncharacterized.
Prunes séchées d'Asie centrale utilisées dans la médecine Unani comme laxatifs, pour la fièvre et comme toniques hépatiques.
Bukhara Plum (Prunus bokhariensis) is a dried fruit from Central Asia traditionally used in Unani medicine as a laxative, antipyretic, and liver tonic. Its primary active compounds include sorbitol, malic acid, phenolic acids, and flavonoids, which contribute to its mild laxative and hepatoprotective effects. Evidence is limited to preliminary studies and traditional use, placing it at an evidence level C.
The laxative effect is primarily due to sorbitol, an osmotic agent that draws water into the intestinal lumen, stimulating peristalsis. Phenolic acids and flavonoids may exert hepatoprotective effects by scavenging free radicals and modulating phase II detoxification enzymes, such as glutathione S-transferase. Antipyretic activity is hypothesized to involve inhibition of prostaglandin synthesis via cyclooxygenase (COX) pathways, though specific receptor interactions remain uncharacterized.