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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Cannabis sativa (hemp)
Un cannabino non psychotrope proven dans des recherches sur la gestion de la douleur et de l'anxiété.
Cannabidiol (CBD) is a non-psychoactive cannabinoid derived from Cannabis sativa (hemp) with evidence-supported analgesic, anxiolytic, anti-epileptic, and anti-inflammatory properties. Its primary active compounds include cannabidiol, minor cannabinoids (CBG, CBC), terpenes, and flavonoids, which collectively contribute to its therapeutic profile.
CBD exerts its effects primarily through modulation of the endocannabinoid system, acting as a negative allosteric modulator of CB1 and CB2 receptors, and inhibiting fatty acid amide hydrolase (FAAH) to increase anandamide levels. It also activates TRPV1 channels, 5-HT1A serotonin receptors, and PPARγ nuclear receptors, contributing to its analgesic, anxiolytic, and anti-inflammatory actions. Additionally, CBD inhibits CYP3A4 and CYP2C19 enzymes, affecting drug metabolism.
Un cannabino non psychotrope proven dans des recherches sur la gestion de la douleur et de l'anxiété.
Cannabidiol (CBD) is a non-psychoactive cannabinoid derived from Cannabis sativa (hemp) with evidence-supported analgesic, anxiolytic, anti-epileptic, and anti-inflammatory properties. Its primary active compounds include cannabidiol, minor cannabinoids (CBG, CBC), terpenes, and flavonoids, which collectively contribute to its therapeutic profile.
CBD exerts its effects primarily through modulation of the endocannabinoid system, acting as a negative allosteric modulator of CB1 and CB2 receptors, and inhibiting fatty acid amide hydrolase (FAAH) to increase anandamide levels. It also activates TRPV1 channels, 5-HT1A serotonin receptors, and PPARγ nuclear receptors, contributing to its analgesic, anxiolytic, and anti-inflammatory actions. Additionally, CBD inhibits CYP3A4 and CYP2C19 enzymes, affecting drug metabolism.