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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Citrus sinensis bioflavonoids
Bioflavonoïdes concentrés utilisés pour les conditions veineuses, l'anti-inflammation et les effets antioxydants, initialement appelés vitamine P.
Citrus bioflavonoids are a group of polyphenolic compounds (including hesperidin, diosmin, rutin, naringenin, and quercetin) derived from citrus fruits, historically termed 'Vitamin P'. They are primarily used for venous conditions such as chronic venous insufficiency and hemorrhoids, as well as for their antioxidant and anti-inflammatory properties. The evidence level is C (limited/pilot studies), though some specific flavonoid combinations have stronger data.
Citrus bioflavonoids exert venotonic effects by inhibiting venous smooth muscle contraction and reducing venous distensibility, possibly through modulation of calcium channels and adrenergic receptors. They also inhibit enzymes involved in inflammation, such as cyclooxygenase (COX) and lipoxygenase (LOX), and reduce capillary permeability by strengthening the endothelial barrier. Additionally, they act as antioxidants by scavenging free radicals and chelating metal ions, and may inhibit platelet aggregation via modulation of thromboxane A2.
Bioflavonoïdes concentrés utilisés pour les conditions veineuses, l'anti-inflammation et les effets antioxydants, initialement appelés vitamine P.
Citrus bioflavonoids are a group of polyphenolic compounds (including hesperidin, diosmin, rutin, naringenin, and quercetin) derived from citrus fruits, historically termed 'Vitamin P'. They are primarily used for venous conditions such as chronic venous insufficiency and hemorrhoids, as well as for their antioxidant and anti-inflammatory properties. The evidence level is C (limited/pilot studies), though some specific flavonoid combinations have stronger data.
Citrus bioflavonoids exert venotonic effects by inhibiting venous smooth muscle contraction and reducing venous distensibility, possibly through modulation of calcium channels and adrenergic receptors. They also inhibit enzymes involved in inflammation, such as cyclooxygenase (COX) and lipoxygenase (LOX), and reduce capillary permeability by strengthening the endothelial barrier. Additionally, they act as antioxidants by scavenging free radicals and chelating metal ions, and may inhibit platelet aggregation via modulation of thromboxane A2.