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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Curcuma caesia
Curcuma indien rare au rhizome teinté de bleu ; utilisé en Ayurveda pour les affections neurologiques et épileptiques.
Curcuma caesia, a rare Indian turmeric with a blue-tinged rhizome, is traditionally used in Ayurveda for neurological conditions such as epilepsy and paralysis. Its primary active compounds include ar-turmerone, alpha-turmerone, camphor, elemenone, and carotol, which contribute to its anticonvulsant, anti-inflammatory, and analgesic properties.
The anticonvulsant activity of Curcuma caesia is primarily attributed to ar-turmerone and camphor, which modulate GABA-A receptors and enhance inhibitory neurotransmission. Its anti-inflammatory effects involve inhibition of cyclooxygenase (COX) and lipoxygenase (LOX) enzymes, reducing prostaglandin and leukotriene synthesis. Additionally, camphor activates TRPV1 and TRPM8 channels, contributing to analgesic and counterirritant actions.
Curcuma indien rare au rhizome teinté de bleu ; utilisé en Ayurveda pour les affections neurologiques et épileptiques.
Curcuma caesia, a rare Indian turmeric with a blue-tinged rhizome, is traditionally used in Ayurveda for neurological conditions such as epilepsy and paralysis. Its primary active compounds include ar-turmerone, alpha-turmerone, camphor, elemenone, and carotol, which contribute to its anticonvulsant, anti-inflammatory, and analgesic properties.
The anticonvulsant activity of Curcuma caesia is primarily attributed to ar-turmerone and camphor, which modulate GABA-A receptors and enhance inhibitory neurotransmission. Its anti-inflammatory effects involve inhibition of cyclooxygenase (COX) and lipoxygenase (LOX) enzymes, reducing prostaglandin and leukotriene synthesis. Additionally, camphor activates TRPV1 and TRPM8 channels, contributing to analgesic and counterirritant actions.