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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Curcuma phaeocaulis
Plante à action de mouvement sanguin en TCM ; rhizome utilisé pour rompre la stase sanguine et traiter les tumeurs.
Curcuma phaeocaulis is a traditional Chinese medicinal herb whose rhizome is used to invigorate blood circulation, break blood stasis, and treat tumors. Its primary modern applications include anti-tumor, anti-inflammatory, analgesic, and antiplatelet effects, supported by limited clinical evidence (Level C). Key active compounds include curzerenone, curcumol, germacrone, furanodiene, guaiol, and beta-elemene.
The herb exhibits anti-tumor activity through induction of apoptosis and cell cycle arrest, mediated by beta-elemene and curcumol which modulate PI3K/Akt and NF-κB pathways. Its anti-inflammatory effects involve inhibition of COX-2 and LOX enzymes, reducing prostaglandin and leukotriene synthesis. Antiplatelet action is attributed to curzerenone and germacrone, which suppress thromboxane A2 formation and platelet aggregation via interference with arachidonic acid metabolism.
Plante à action de mouvement sanguin en TCM ; rhizome utilisé pour rompre la stase sanguine et traiter les tumeurs.
Curcuma phaeocaulis is a traditional Chinese medicinal herb whose rhizome is used to invigorate blood circulation, break blood stasis, and treat tumors. Its primary modern applications include anti-tumor, anti-inflammatory, analgesic, and antiplatelet effects, supported by limited clinical evidence (Level C). Key active compounds include curzerenone, curcumol, germacrone, furanodiene, guaiol, and beta-elemene.
The herb exhibits anti-tumor activity through induction of apoptosis and cell cycle arrest, mediated by beta-elemene and curcumol which modulate PI3K/Akt and NF-κB pathways. Its anti-inflammatory effects involve inhibition of COX-2 and LOX enzymes, reducing prostaglandin and leukotriene synthesis. Antiplatelet action is attributed to curzerenone and germacrone, which suppress thromboxane A2 formation and platelet aggregation via interference with arachidonic acid metabolism.