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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Gynura pseudochina
Herbe jamu avec une réputation croissante pour des propriétés anti-tumeur, مؤازرة à la pression artérielle et guérison de plaies.
Daun Dewa (Gynura pseudochina) is a Jamu herb traditionally used for hypertension, wound healing, and tumor prevention. It contains alkaloids, flavonoids, tannins, and sterols, with limited evidence (C) suggesting antihypertensive, anticoagulant, and anti-tumor activities. Key active compounds include pyrrolizidine alkaloids, which raise hepatotoxicity concerns.
The antihypertensive effect may involve inhibition of angiotensin-converting enzyme (ACE) and calcium channel blockade by flavonoids. Wound healing is attributed to tannins and sterols promoting collagen synthesis and angiogenesis. Anti-tumor activity may be due to alkaloid-induced apoptosis and cell cycle arrest via p53 pathway activation. Anticoagulant effects likely stem from inhibition of platelet aggregation and vitamin K antagonism.
Herbe jamu avec une réputation croissante pour des propriétés anti-tumeur, مؤازرة à la pression artérielle et guérison de plaies.
Daun Dewa (Gynura pseudochina) is a Jamu herb traditionally used for hypertension, wound healing, and tumor prevention. It contains alkaloids, flavonoids, tannins, and sterols, with limited evidence (C) suggesting antihypertensive, anticoagulant, and anti-tumor activities. Key active compounds include pyrrolizidine alkaloids, which raise hepatotoxicity concerns.
The antihypertensive effect may involve inhibition of angiotensin-converting enzyme (ACE) and calcium channel blockade by flavonoids. Wound healing is attributed to tannins and sterols promoting collagen synthesis and angiogenesis. Anti-tumor activity may be due to alkaloid-induced apoptosis and cell cycle arrest via p53 pathway activation. Anticoagulant effects likely stem from inhibition of platelet aggregation and vitamin K antagonism.