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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Taxillus sutchuenensis
Parasite de gui utilisé en médecine traditionnelle chinoise pour tonifier le foie et les reins, renforcer les os, calmer le fœtus et abaisser la pression artérielle.
Dian Ji Sheng (Taxillus sutchuenensis) is a hemiparasitic plant used in Traditional Chinese Medicine to tonify the Liver and Kidney, strengthen bones, calm the fetus, and lower blood pressure. It is commonly employed for musculoskeletal weakness, threatened miscarriage, hypertension, and liver deficiency. Key active compounds include quercetin, avicularin, hyperoside, oleanolic acid, and phytol.
The antihypertensive effect is attributed to quercetin and hyperoside, which promote vasodilation via nitric oxide (NO)/cGMP pathway and inhibit angiotensin-converting enzyme (ACE). Anti-inflammatory activity involves suppression of NF-κB and COX-2 pathways, while immunomodulation is mediated through oleanolic acid and avicularin, which modulate cytokine production and T-cell activity. Fetal support is traditionally linked to uterine relaxation and improved placental circulation, possibly via calcium channel blockade.
Parasite de gui utilisé en médecine traditionnelle chinoise pour tonifier le foie et les reins, renforcer les os, calmer le fœtus et abaisser la pression artérielle.
Dian Ji Sheng (Taxillus sutchuenensis) is a hemiparasitic plant used in Traditional Chinese Medicine to tonify the Liver and Kidney, strengthen bones, calm the fetus, and lower blood pressure. It is commonly employed for musculoskeletal weakness, threatened miscarriage, hypertension, and liver deficiency. Key active compounds include quercetin, avicularin, hyperoside, oleanolic acid, and phytol.
The antihypertensive effect is attributed to quercetin and hyperoside, which promote vasodilation via nitric oxide (NO)/cGMP pathway and inhibit angiotensin-converting enzyme (ACE). Anti-inflammatory activity involves suppression of NF-κB and COX-2 pathways, while immunomodulation is mediated through oleanolic acid and avicularin, which modulate cytokine production and T-cell activity. Fetal support is traditionally linked to uterine relaxation and improved placental circulation, possibly via calcium channel blockade.