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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Digitalis lanata
Digitale à feuilles laineuses originaire du sud-est de l'Europe, utilisée comme source principale de digoxine pour le traitement de l'insuffisance cardiaque.
Digitalis lanata (woolly foxglove) is a European herb historically used for heart failure and edema, now primarily valued as the commercial source of digoxin. Its key active compounds include the cardiac glycosides digoxin, digitoxin, and lanatoside C. Modern clinical use is restricted to purified digoxin due to the plant's narrow therapeutic index and high toxicity risk.
Cardiac glycosides from Digitalis lanata, such as digoxin, inhibit the Na+/K+-ATPase pump in cardiac myocytes, leading to increased intracellular sodium. This reduces calcium efflux via the Na+/Ca2+ exchanger, raising intracellular calcium and enhancing myocardial contractility (positive inotropy). Additionally, digoxin increases vagal tone, slowing atrioventricular conduction and reducing heart rate, which is beneficial in atrial fibrillation. The drug also has direct electrophysiological effects on the heart, prolonging the refractory period.
Digitale à feuilles laineuses originaire du sud-est de l'Europe, utilisée comme source principale de digoxine pour le traitement de l'insuffisance cardiaque.
Digitalis lanata (woolly foxglove) is a European herb historically used for heart failure and edema, now primarily valued as the commercial source of digoxin. Its key active compounds include the cardiac glycosides digoxin, digitoxin, and lanatoside C. Modern clinical use is restricted to purified digoxin due to the plant's narrow therapeutic index and high toxicity risk.
Cardiac glycosides from Digitalis lanata, such as digoxin, inhibit the Na+/K+-ATPase pump in cardiac myocytes, leading to increased intracellular sodium. This reduces calcium efflux via the Na+/Ca2+ exchanger, raising intracellular calcium and enhancing myocardial contractility (positive inotropy). Additionally, digoxin increases vagal tone, slowing atrioventricular conduction and reducing heart rate, which is beneficial in atrial fibrillation. The drug also has direct electrophysiological effects on the heart, prolonging the refractory period.