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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Eryngium foetidum
Apiaceae des Amériques tropicales et des Caraïbes à odeur forte de coriandre; utilisée medicinalement pour la fièvre, les convulsions et les plaintes digestives.
Eryngium foetidum, also known as culantro or fitweed, is a tropical herb in the Apiaceae family with a strong coriander-like aroma. It is traditionally used in Caribbean and Latin American folk medicine for its antipyretic, anticonvulsant, and digestive properties. Key active compounds include eryngial, flavonoids, triterpenoids, and saponins.
The antipyretic effect is attributed to inhibition of prostaglandin synthesis via COX enzyme modulation, similar to other Apiaceae species. Anticonvulsant activity is primarily linked to eryngial, which potentiates GABA-A receptor-mediated chloride influx, reducing neuronal excitability. Flavonoids and saponins contribute to antimicrobial effects by disrupting microbial cell membranes and inhibiting efflux pumps. Triterpenoids may exert anti-inflammatory actions through suppression of NF-κB pathway.
Apiaceae des Amériques tropicales et des Caraïbes à odeur forte de coriandre; utilisée medicinalement pour la fièvre, les convulsions et les plaintes digestives.
Eryngium foetidum, also known as culantro or fitweed, is a tropical herb in the Apiaceae family with a strong coriander-like aroma. It is traditionally used in Caribbean and Latin American folk medicine for its antipyretic, anticonvulsant, and digestive properties. Key active compounds include eryngial, flavonoids, triterpenoids, and saponins.
The antipyretic effect is attributed to inhibition of prostaglandin synthesis via COX enzyme modulation, similar to other Apiaceae species. Anticonvulsant activity is primarily linked to eryngial, which potentiates GABA-A receptor-mediated chloride influx, reducing neuronal excitability. Flavonoids and saponins contribute to antimicrobial effects by disrupting microbial cell membranes and inhibiting efflux pumps. Triterpenoids may exert anti-inflammatory actions through suppression of NF-κB pathway.