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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Frangula alnus
A European shrub with laxative anthraquinone bark used for constipation.
Frangula alnus (alder buckthorn) is a European shrub whose aged bark contains anthraquinone glycosides such as frangulin and glucofrangulin, which act as stimulant laxatives. It is primarily used for short-term relief of constipation and to promote bowel regularity, with additional traditional applications as a liver tonic and digestive aid. The herb also contains emodin and tannins, contributing to its purgative and mild astringent properties.
The anthraquinone glycosides (frangulin, glucofrangulin) are hydrolyzed by colonic bacteria to active aglycones like emodin, which stimulate peristalsis by increasing colonic motility and fluid secretion. These compounds act on enteric neurons and may involve prostaglandin E2 release, while also inhibiting Na+/K+-ATPase in the intestinal epithelium, reducing water absorption. The laxative effect typically occurs 6–12 hours after oral administration.
A European shrub with laxative anthraquinone bark used for constipation.
Frangula alnus (alder buckthorn) is a European shrub whose aged bark contains anthraquinone glycosides such as frangulin and glucofrangulin, which act as stimulant laxatives. It is primarily used for short-term relief of constipation and to promote bowel regularity, with additional traditional applications as a liver tonic and digestive aid. The herb also contains emodin and tannins, contributing to its purgative and mild astringent properties.
The anthraquinone glycosides (frangulin, glucofrangulin) are hydrolyzed by colonic bacteria to active aglycones like emodin, which stimulate peristalsis by increasing colonic motility and fluid secretion. These compounds act on enteric neurons and may involve prostaglandin E2 release, while also inhibiting Na+/K+-ATPase in the intestinal epithelium, reducing water absorption. The laxative effect typically occurs 6–12 hours after oral administration.