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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Ganoderma tsugae
A North American Reishi relative growing on hemlock trees, used for immune support.
Ganoderma tsugae, a North American reishi relative growing on hemlock trees, is used for immune support, liver protection, and as an adjunct in cancer therapy. Its primary active compounds include ganoderic acids, polysaccharides, triterpenoids, and ergosterol, which contribute to its immunomodulatory, hepatoprotective, anti-tumor, and anti-inflammatory effects.
Ganoderic acids and triterpenoids inhibit NF-κB and COX-2 pathways, reducing pro-inflammatory cytokines (TNF-α, IL-6) and prostaglandin synthesis. Polysaccharides, particularly β-glucans, activate macrophages and dendritic cells via dectin-1 and TLR-2/4 receptors, enhancing innate immunity. Ergosterol and its derivatives modulate Th1/Th2 balance and induce apoptosis in cancer cells through caspase-3 activation and mitochondrial pathway disruption. Hepatoprotective effects are mediated by upregulation of Nrf2 and antioxidant enzymes (SOD, catalase), reducing oxidative stress and liver enzyme leakage.
A North American Reishi relative growing on hemlock trees, used for immune support.
Ganoderma tsugae, a North American reishi relative growing on hemlock trees, is used for immune support, liver protection, and as an adjunct in cancer therapy. Its primary active compounds include ganoderic acids, polysaccharides, triterpenoids, and ergosterol, which contribute to its immunomodulatory, hepatoprotective, anti-tumor, and anti-inflammatory effects.
Ganoderic acids and triterpenoids inhibit NF-κB and COX-2 pathways, reducing pro-inflammatory cytokines (TNF-α, IL-6) and prostaglandin synthesis. Polysaccharides, particularly β-glucans, activate macrophages and dendritic cells via dectin-1 and TLR-2/4 receptors, enhancing innate immunity. Ergosterol and its derivatives modulate Th1/Th2 balance and induce apoptosis in cancer cells through caspase-3 activation and mitochondrial pathway disruption. Hepatoprotective effects are mediated by upregulation of Nrf2 and antioxidant enzymes (SOD, catalase), reducing oxidative stress and liver enzyme leakage.