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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Lithospermum officinale
Perennial of European roadsides; seeds used in folk medicine as diuretic; roots studied for contraceptive properties and anti-gonadotropic effects.
Gromwell (Lithospermum officinale) is a perennial herb traditionally used as a diuretic and contraceptive, with modern research focusing on its anti-gonadotropic and antithyroid properties. Key active compounds include lithospermic acid, phenolics, tannins, and silicic acid, which contribute to its pharmacological effects.
Lithospermic acid inhibits gonadotropin release by interfering with hypothalamic-pituitary signaling, reducing luteinizing hormone and follicle-stimulating hormone levels. The antithyroid action is mediated through inhibition of thyroid peroxidase and reduced thyroxine synthesis, likely due to phenolic compounds. Diuretic effects may result from increased renal blood flow and inhibition of sodium reabsorption via tannins and silicic acid.
Perennial of European roadsides; seeds used in folk medicine as diuretic; roots studied for contraceptive properties and anti-gonadotropic effects.
Gromwell (Lithospermum officinale) is a perennial herb traditionally used as a diuretic and contraceptive, with modern research focusing on its anti-gonadotropic and antithyroid properties. Key active compounds include lithospermic acid, phenolics, tannins, and silicic acid, which contribute to its pharmacological effects.
Lithospermic acid inhibits gonadotropin release by interfering with hypothalamic-pituitary signaling, reducing luteinizing hormone and follicle-stimulating hormone levels. The antithyroid action is mediated through inhibition of thyroid peroxidase and reduced thyroxine synthesis, likely due to phenolic compounds. Diuretic effects may result from increased renal blood flow and inhibition of sodium reabsorption via tannins and silicic acid.