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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Piper kadsura
A TCM vine that warms the meridians, dispels wind-damp, and unblocks the collaterals; used for rheumatoid arthritis and cold-type joint pain.
Hai Feng Teng (Piper kadsura) is a traditional Chinese medicinal vine used to warm the meridians, dispel wind-damp, and unblock collaterals, primarily for rheumatoid arthritis and cold-type joint pain. Its active compounds include lignans, piperine, kadsurene, and flavonoids, which contribute to its anti-inflammatory, analgesic, and antispasmodic properties. Evidence level C indicates limited clinical data, with most support from traditional use and preliminary studies.
The herb's analgesic and anti-inflammatory effects are mediated through activation of TRPV1 receptors by piperine, leading to desensitization of pain pathways. Lignans and flavonoids inhibit cyclooxygenase (COX) and lipoxygenase (LOX) enzymes, reducing prostaglandin and leukotriene synthesis. Additionally, kadsurene modulates NF-κB signaling, decreasing pro-inflammatory cytokines such as TNF-α and IL-6. The warming property is attributed to enhanced local blood flow via vasodilation and stimulation of transient receptor potential channels.
A TCM vine that warms the meridians, dispels wind-damp, and unblocks the collaterals; used for rheumatoid arthritis and cold-type joint pain.
Hai Feng Teng (Piper kadsura) is a traditional Chinese medicinal vine used to warm the meridians, dispel wind-damp, and unblock collaterals, primarily for rheumatoid arthritis and cold-type joint pain. Its active compounds include lignans, piperine, kadsurene, and flavonoids, which contribute to its anti-inflammatory, analgesic, and antispasmodic properties. Evidence level C indicates limited clinical data, with most support from traditional use and preliminary studies.
The herb's analgesic and anti-inflammatory effects are mediated through activation of TRPV1 receptors by piperine, leading to desensitization of pain pathways. Lignans and flavonoids inhibit cyclooxygenase (COX) and lipoxygenase (LOX) enzymes, reducing prostaglandin and leukotriene synthesis. Additionally, kadsurene modulates NF-κB signaling, decreasing pro-inflammatory cytokines such as TNF-α and IL-6. The warming property is attributed to enhanced local blood flow via vasodilation and stimulation of transient receptor potential channels.