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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Nyctanthes arbor-tristis
Indian tree with flowers and leaves used in Ayurveda and folk medicine for fever and rheumatism.
Nyctanthes arbor-tristis (Harshingar) is a sacred Ayurvedic tree whose leaves and flowers are traditionally used for malaria, rheumatism, and fever. Its primary active compounds include nyctanthoside, iridoid glycosides, and beta-sitosterol, which contribute to anti-inflammatory, antimalarial, and antiviral properties. Modern research suggests potential in these areas, though clinical evidence remains limited (Grade C).
The iridoid glycosides, particularly nyctanthoside, inhibit cyclooxygenase-2 (COX-2) and nuclear factor-kappa B (NF-κB) pathways, reducing prostaglandin synthesis and pro-inflammatory cytokines. Beta-sitosterol modulates immune responses and may suppress joint inflammation by inhibiting leukotriene formation. Antimalarial activity is attributed to interference with Plasmodium falciparum heme detoxification, while flavonoids and tannins contribute to antioxidant and antiviral effects by scavenging free radicals and inhibiting viral replication.
Indian tree with flowers and leaves used in Ayurveda and folk medicine for fever and rheumatism.
Nyctanthes arbor-tristis (Harshingar) is a sacred Ayurvedic tree whose leaves and flowers are traditionally used for malaria, rheumatism, and fever. Its primary active compounds include nyctanthoside, iridoid glycosides, and beta-sitosterol, which contribute to anti-inflammatory, antimalarial, and antiviral properties. Modern research suggests potential in these areas, though clinical evidence remains limited (Grade C).
The iridoid glycosides, particularly nyctanthoside, inhibit cyclooxygenase-2 (COX-2) and nuclear factor-kappa B (NF-κB) pathways, reducing prostaglandin synthesis and pro-inflammatory cytokines. Beta-sitosterol modulates immune responses and may suppress joint inflammation by inhibiting leukotriene formation. Antimalarial activity is attributed to interference with Plasmodium falciparum heme detoxification, while flavonoids and tannins contribute to antioxidant and antiviral effects by scavenging free radicals and inhibiting viral replication.