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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Hedera helix
A European climbing plant with ivy leaf extract well-studied for bronchitis and respiratory conditions.
Hedera helix (ivy leaf) is a climbing plant native to Europe whose leaf extracts are standardized for use in respiratory conditions. It is primarily employed as an expectorant and antispasmodic for bronchitis management, with key active compounds including α-hederin, hederasaponin C, flavonoids, and caffeic acid. Evidence from clinical studies supports its efficacy in reducing cough frequency and severity.
The saponins α-hederin and hederasaponin C exert secretolytic effects by stimulating β₂-adrenergic receptors on bronchial epithelial cells, increasing mucus production and reducing viscosity. Additionally, they exhibit antispasmodic activity through inhibition of phosphodiesterase and modulation of calcium channels, leading to bronchodilation. Anti-inflammatory actions involve suppression of NF-κB and reduction of pro-inflammatory cytokines such as IL-8 and TNF-α.
A European climbing plant with ivy leaf extract well-studied for bronchitis and respiratory conditions.
Hedera helix (ivy leaf) is a climbing plant native to Europe whose leaf extracts are standardized for use in respiratory conditions. It is primarily employed as an expectorant and antispasmodic for bronchitis management, with key active compounds including α-hederin, hederasaponin C, flavonoids, and caffeic acid. Evidence from clinical studies supports its efficacy in reducing cough frequency and severity.
The saponins α-hederin and hederasaponin C exert secretolytic effects by stimulating β₂-adrenergic receptors on bronchial epithelial cells, increasing mucus production and reducing viscosity. Additionally, they exhibit antispasmodic activity through inhibition of phosphodiesterase and modulation of calcium channels, leading to bronchodilation. Anti-inflammatory actions involve suppression of NF-κB and reduction of pro-inflammatory cytokines such as IL-8 and TNF-α.