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Hedyotis diffusa
Major TCM cancer-support herb that clears heat and fire toxin, promotes urination, and dispels blood stasis. One of the most commonly used herbs in traditional Chinese cancer formulas.
Hedyotis diffusa (Hedyotis Herb) is a traditional Chinese medicinal herb widely used in cancer-support formulas for its heat-clearing, toxin-removing, and blood-cooling properties. Its primary active compounds include ursolic acid, oleanolic acid, asperuloside, and various iridoids, which contribute to its antitumor, anti-inflammatory, and antioxidant effects. Evidence is limited to preclinical and pilot clinical studies (Level C), supporting its role as an adjunct in cancer therapy and for inflammatory conditions.
Hedyotis diffusa exhibits antitumor activity through multiple mechanisms: induction of apoptosis via mitochondrial pathway (upregulation of Bax, downregulation of Bcl-2), inhibition of angiogenesis by suppressing VEGF and HIF-1α, and modulation of immune response via activation of natural killer cells and macrophages. Its anti-inflammatory effects are mediated by inhibition of COX-2 and LOX enzymes, reducing prostaglandin and leukotriene synthesis. The iridoid glycosides and triterpenoids also scavenge free radicals and inhibit NF-κB signaling, contributing to its antioxidant and anti-inflammatory profile.
Major TCM cancer-support herb that clears heat and fire toxin, promotes urination, and dispels blood stasis. One of the most commonly used herbs in traditional Chinese cancer formulas.
Hedyotis diffusa (Hedyotis Herb) is a traditional Chinese medicinal herb widely used in cancer-support formulas for its heat-clearing, toxin-removing, and blood-cooling properties. Its primary active compounds include ursolic acid, oleanolic acid, asperuloside, and various iridoids, which contribute to its antitumor, anti-inflammatory, and antioxidant effects. Evidence is limited to preclinical and pilot clinical studies (Level C), supporting its role as an adjunct in cancer therapy and for inflammatory conditions.
Hedyotis diffusa exhibits antitumor activity through multiple mechanisms: induction of apoptosis via mitochondrial pathway (upregulation of Bax, downregulation of Bcl-2), inhibition of angiogenesis by suppressing VEGF and HIF-1α, and modulation of immune response via activation of natural killer cells and macrophages. Its anti-inflammatory effects are mediated by inhibition of COX-2 and LOX enzymes, reducing prostaglandin and leukotriene synthesis. The iridoid glycosides and triterpenoids also scavenge free radicals and inhibit NF-κB signaling, contributing to its antioxidant and anti-inflammatory profile.