Loading...
Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
Loading...
Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Picrasma quassioides
A Tibetan bitter herb used in Sowa Rigpa to clear heat, dry dampness, and treat skin conditions.
Himalayan Neem (Picrasma quassioides) is a bitter herb from the Simaroubaceae family, traditionally used in Sowa Rigpa to clear heat, dry dampness, and treat skin infections. Modern research supports its antimicrobial, antiprotozoal, and anti-inflammatory activities, attributed to picrasidine alkaloids, quassinoids, and beta-carboline alkaloids. It is primarily employed for skin conditions, fever, liver heat, and digestive issues.
Quassinoids inhibit protein synthesis in microbial cells by binding to the 60S ribosomal subunit, while beta-carboline alkaloids modulate GABA-A receptors, contributing to sedative and antiprotozoal effects. Picrasidine alkaloids suppress NF-κB and COX-2 pathways, reducing pro-inflammatory cytokine production. Additionally, bitter principles stimulate gastric acid secretion and bile flow, aiding digestion.
A Tibetan bitter herb used in Sowa Rigpa to clear heat, dry dampness, and treat skin conditions.
Himalayan Neem (Picrasma quassioides) is a bitter herb from the Simaroubaceae family, traditionally used in Sowa Rigpa to clear heat, dry dampness, and treat skin infections. Modern research supports its antimicrobial, antiprotozoal, and anti-inflammatory activities, attributed to picrasidine alkaloids, quassinoids, and beta-carboline alkaloids. It is primarily employed for skin conditions, fever, liver heat, and digestive issues.
Quassinoids inhibit protein synthesis in microbial cells by binding to the 60S ribosomal subunit, while beta-carboline alkaloids modulate GABA-A receptors, contributing to sedative and antiprotozoal effects. Picrasidine alkaloids suppress NF-κB and COX-2 pathways, reducing pro-inflammatory cytokine production. Additionally, bitter principles stimulate gastric acid secretion and bile flow, aiding digestion.