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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Kaempferia elegans
Ornamental Thai ginger with folk use for digestive conditions and as topical anti-inflammatory.
Kaempferia elegans is an ornamental ginger used in Thai folk medicine for digestive conditions and as a topical anti-inflammatory. Modern research indicates anti-inflammatory and antimicrobial activities, attributed to flavonoids, terpenes, and phenolic compounds. Its traditional uses are supported by preliminary evidence, but clinical data remain limited.
The anti-inflammatory effects are mediated through inhibition of cyclooxygenase-2 (COX-2) and 5-lipoxygenase (5-LOX) pathways, reducing prostaglandin and leukotriene synthesis. Antimicrobial activity involves disruption of bacterial cell membranes and inhibition of biofilm formation, likely due to terpenoid and phenolic constituents. Flavonoids may also modulate nuclear factor-kappa B (NF-κB) signaling, decreasing pro-inflammatory cytokine production.
Ornamental Thai ginger with folk use for digestive conditions and as topical anti-inflammatory.
Kaempferia elegans is an ornamental ginger used in Thai folk medicine for digestive conditions and as a topical anti-inflammatory. Modern research indicates anti-inflammatory and antimicrobial activities, attributed to flavonoids, terpenes, and phenolic compounds. Its traditional uses are supported by preliminary evidence, but clinical data remain limited.
The anti-inflammatory effects are mediated through inhibition of cyclooxygenase-2 (COX-2) and 5-lipoxygenase (5-LOX) pathways, reducing prostaglandin and leukotriene synthesis. Antimicrobial activity involves disruption of bacterial cell membranes and inhibition of biofilm formation, likely due to terpenoid and phenolic constituents. Flavonoids may also modulate nuclear factor-kappa B (NF-κB) signaling, decreasing pro-inflammatory cytokine production.