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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Kaempferia galanga
Low-growing aromatic ginger from Southeast Asia used in Jamu Indonesian medicine and throughout the region for digestion and pain.
Kaempferia galanga (kencur) is an aromatic rhizome from Southeast Asia, traditionally used in Indonesian Jamu medicine for digestive complaints and joint pain. Its primary active compounds include methyl cinnamate, camphor, borneol, and ethyl p-methoxycinnamate, which contribute to its analgesic, anti-inflammatory, and carminative properties.
Methyl cinnamate and ethyl p-methoxycinnamate inhibit cyclooxygenase (COX) enzymes and modulate transient receptor potential (TRP) channels, particularly TRPV1, reducing pain and inflammation. Camphor and borneol act as counterirritants and local anesthetics via sodium channel blockade and TRPM8 activation. The carminative effect is attributed to spasmolytic activity on gastrointestinal smooth muscle, likely mediated by calcium channel antagonism and nitric oxide release.
Low-growing aromatic ginger from Southeast Asia used in Jamu Indonesian medicine and throughout the region for digestion and pain.
Kaempferia galanga (kencur) is an aromatic rhizome from Southeast Asia, traditionally used in Indonesian Jamu medicine for digestive complaints and joint pain. Its primary active compounds include methyl cinnamate, camphor, borneol, and ethyl p-methoxycinnamate, which contribute to its analgesic, anti-inflammatory, and carminative properties.
Methyl cinnamate and ethyl p-methoxycinnamate inhibit cyclooxygenase (COX) enzymes and modulate transient receptor potential (TRP) channels, particularly TRPV1, reducing pain and inflammation. Camphor and borneol act as counterirritants and local anesthetics via sodium channel blockade and TRPM8 activation. The carminative effect is attributed to spasmolytic activity on gastrointestinal smooth muscle, likely mediated by calcium channel antagonism and nitric oxide release.