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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Cinnamomum burmanii
Indonesian cinnamon variety used in Jamu for diabetes management, digestive support, and warming the body.
Kayu Manis Jamu (Cinnamomum burmanii) is a cinnamon variety native to Indonesia, traditionally used in Jamu for diabetes management, digestive support, and warming the body. Its primary active compounds include cinnamaldehyde, coumarin, procyanidins, and eugenol, which contribute to its hypoglycemic, antimicrobial, and anti-inflammatory properties. Evidence level C indicates limited clinical studies supporting its modern uses.
Cinnamaldehyde activates TRPA1 channels, enhancing insulin sensitivity and glucose uptake in adipocytes and skeletal muscle. It also inhibits alpha-glucosidase and aldose reductase, reducing postprandial hyperglycemia. Procyanidins exert antioxidant and anti-inflammatory effects via NF-κB pathway modulation. Coumarin, at high doses, is hepatotoxic due to CYP450-mediated metabolism to 3,4-coumarin epoxide, causing liver injury.
Indonesian cinnamon variety used in Jamu for diabetes management, digestive support, and warming the body.
Kayu Manis Jamu (Cinnamomum burmanii) is a cinnamon variety native to Indonesia, traditionally used in Jamu for diabetes management, digestive support, and warming the body. Its primary active compounds include cinnamaldehyde, coumarin, procyanidins, and eugenol, which contribute to its hypoglycemic, antimicrobial, and anti-inflammatory properties. Evidence level C indicates limited clinical studies supporting its modern uses.
Cinnamaldehyde activates TRPA1 channels, enhancing insulin sensitivity and glucose uptake in adipocytes and skeletal muscle. It also inhibits alpha-glucosidase and aldose reductase, reducing postprandial hyperglycemia. Procyanidins exert antioxidant and anti-inflammatory effects via NF-κB pathway modulation. Coumarin, at high doses, is hepatotoxic due to CYP450-mediated metabolism to 3,4-coumarin epoxide, causing liver injury.