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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Tinospora sinensis
Tibetan and Chinese climbing vine used in Sowa Rigpa and TCM for fevers, immune support, and as bitter tonic adaptogen.
Tinospora sinensis, known as Kheemp Tibetan, is a climbing vine used in Sowa Rigpa and Traditional Chinese Medicine. It is primarily employed as an antipyretic, immunomodulatory, and adaptogenic bitter tonic, with active compounds including tinosporin, berberine, palmatine, and magnoflorine. Modern research supports its hepatoprotective and immune-supporting properties, though clinical evidence remains limited.
The immunomodulatory effects are mediated through activation of macrophages and modulation of cytokine production (e.g., TNF-α, IL-6). Antipyretic action involves inhibition of prostaglandin synthesis via COX-2 suppression. Hepatoprotection is attributed to antioxidant activity (scavenging free radicals) and upregulation of phase II detoxification enzymes. Adaptogenic properties are linked to modulation of the hypothalamic-pituitary-adrenal (HPA) axis and stress-induced cortisol release.
Tibetan and Chinese climbing vine used in Sowa Rigpa and TCM for fevers, immune support, and as bitter tonic adaptogen.
Tinospora sinensis, known as Kheemp Tibetan, is a climbing vine used in Sowa Rigpa and Traditional Chinese Medicine. It is primarily employed as an antipyretic, immunomodulatory, and adaptogenic bitter tonic, with active compounds including tinosporin, berberine, palmatine, and magnoflorine. Modern research supports its hepatoprotective and immune-supporting properties, though clinical evidence remains limited.
The immunomodulatory effects are mediated through activation of macrophages and modulation of cytokine production (e.g., TNF-α, IL-6). Antipyretic action involves inhibition of prostaglandin synthesis via COX-2 suppression. Hepatoprotection is attributed to antioxidant activity (scavenging free radicals) and upregulation of phase II detoxification enzymes. Adaptogenic properties are linked to modulation of the hypothalamic-pituitary-adrenal (HPA) axis and stress-induced cortisol release.