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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Liquidambar styraciflua
North American tree producing fragrant storax balsam resin used by Native peoples for skin conditions, respiratory complaints, and wounds.
Liquidambar styraciflua, a North American tree, yields a fragrant storax balsam resin traditionally used by Native Americans for wound healing, skin conditions, and respiratory complaints. Modern evidence (Level C) supports its topical antimicrobial and expectorant properties, attributed to active compounds such as styracin, cinnamyl cinnamate, vanillin, and cinnamic acid.
The resin's antimicrobial action is primarily due to cinnamic acid and vanillin, which disrupt bacterial cell membranes and inhibit microbial enzyme activity. Expectorant effects are mediated by cinnamyl cinnamate, which stimulates bronchial secretion via TRPA1 receptor activation, enhancing mucociliary clearance. Anti-inflammatory activity involves inhibition of cyclooxygenase (COX) and lipoxygenase (LOX) pathways, reducing prostaglandin and leukotriene synthesis. Wound healing is promoted through increased fibroblast proliferation and collagen deposition, likely via vanillin-induced TGF-β signaling.
North American tree producing fragrant storax balsam resin used by Native peoples for skin conditions, respiratory complaints, and wounds.
Liquidambar styraciflua, a North American tree, yields a fragrant storax balsam resin traditionally used by Native Americans for wound healing, skin conditions, and respiratory complaints. Modern evidence (Level C) supports its topical antimicrobial and expectorant properties, attributed to active compounds such as styracin, cinnamyl cinnamate, vanillin, and cinnamic acid.
The resin's antimicrobial action is primarily due to cinnamic acid and vanillin, which disrupt bacterial cell membranes and inhibit microbial enzyme activity. Expectorant effects are mediated by cinnamyl cinnamate, which stimulates bronchial secretion via TRPA1 receptor activation, enhancing mucociliary clearance. Anti-inflammatory activity involves inhibition of cyclooxygenase (COX) and lipoxygenase (LOX) pathways, reducing prostaglandin and leukotriene synthesis. Wound healing is promoted through increased fibroblast proliferation and collagen deposition, likely via vanillin-induced TGF-β signaling.