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Lonicera japonica
TCM herb used to clear heat and toxins; one of the most commonly used herbs for fever, infections, and inflammatory conditions across East Asian medicine.
Lonicera japonica (Japanese honeysuckle) is a widely used herb in Traditional Chinese Medicine for clearing heat and toxins, primarily indicated for febrile illnesses, respiratory infections, and inflammatory skin conditions. Its key active compounds include chlorogenic acid, luteolin, caffeic acid, quercetin, and lonicerosides, which contribute to its antibacterial, antiviral, anti-inflammatory, and antioxidant properties.
The herb exerts antibacterial effects via disruption of bacterial cell wall synthesis and inhibition of biofilm formation, particularly through chlorogenic acid and luteolin. Antiviral activity involves inhibition of viral neuraminidase and hemagglutinin, as well as modulation of host immune responses via NF-κB and MAPK pathways. Anti-inflammatory actions are mediated by suppression of COX-2, iNOS, and pro-inflammatory cytokines (e.g., TNF-α, IL-6) through activation of Nrf2 and inhibition of TLR4 signaling. Antioxidant effects are attributed to direct free radical scavenging by flavonoids and phenolic acids.
TCM herb used to clear heat and toxins; one of the most commonly used herbs for fever, infections, and inflammatory conditions across East Asian medicine.
Lonicera japonica (Japanese honeysuckle) is a widely used herb in Traditional Chinese Medicine for clearing heat and toxins, primarily indicated for febrile illnesses, respiratory infections, and inflammatory skin conditions. Its key active compounds include chlorogenic acid, luteolin, caffeic acid, quercetin, and lonicerosides, which contribute to its antibacterial, antiviral, anti-inflammatory, and antioxidant properties.
The herb exerts antibacterial effects via disruption of bacterial cell wall synthesis and inhibition of biofilm formation, particularly through chlorogenic acid and luteolin. Antiviral activity involves inhibition of viral neuraminidase and hemagglutinin, as well as modulation of host immune responses via NF-κB and MAPK pathways. Anti-inflammatory actions are mediated by suppression of COX-2, iNOS, and pro-inflammatory cytokines (e.g., TNF-α, IL-6) through activation of Nrf2 and inhibition of TLR4 signaling. Antioxidant effects are attributed to direct free radical scavenging by flavonoids and phenolic acids.