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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Ilex pubescens
TCM herb used for cardiovascular conditions, particularly angina, thrombotic stroke, and blood stasis in the heart and vessels.
Mao Dong Qing (Ilex pubescens) is a traditional Chinese herb used primarily for cardiovascular conditions such as angina and thrombotic stroke. Its key active compounds include ilexsaponins, pedunculoside, ursolic acid, flavonoids, and phenolic acids, which contribute to its anticoagulant, vasodilatory, anti-inflammatory, and cardioprotective effects.
The herb exhibits anticoagulant activity through inhibition of platelet aggregation and modulation of coagulation factors, likely mediated by saponins and phenolic acids. Vasodilation is achieved via endothelial nitric oxide synthase (eNOS) activation and calcium channel blockade. Anti-inflammatory effects involve suppression of the NF-κB pathway and reduction of pro-inflammatory cytokines. Cardioprotection may be due to antioxidant activity and improvement of myocardial energy metabolism.
TCM herb used for cardiovascular conditions, particularly angina, thrombotic stroke, and blood stasis in the heart and vessels.
Mao Dong Qing (Ilex pubescens) is a traditional Chinese herb used primarily for cardiovascular conditions such as angina and thrombotic stroke. Its key active compounds include ilexsaponins, pedunculoside, ursolic acid, flavonoids, and phenolic acids, which contribute to its anticoagulant, vasodilatory, anti-inflammatory, and cardioprotective effects.
The herb exhibits anticoagulant activity through inhibition of platelet aggregation and modulation of coagulation factors, likely mediated by saponins and phenolic acids. Vasodilation is achieved via endothelial nitric oxide synthase (eNOS) activation and calcium channel blockade. Anti-inflammatory effects involve suppression of the NF-κB pathway and reduction of pro-inflammatory cytokines. Cardioprotection may be due to antioxidant activity and improvement of myocardial energy metabolism.