Loading...
Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
Loading...
Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Mikania micrantha
Invasive tropical Asteraceae used widely in traditional medicine across Asia and Pacific for wound healing and fever.
Mikania micrantha, an invasive tropical Asteraceae, is traditionally used across Asia and the Pacific for wound healing, fever, and malaria management. Its primary active compounds include sesquiterpene lactones (mikanolide, dihydromikanolide) and flavonoids, which contribute to antimicrobial, anti-inflammatory, and wound-healing properties. Evidence is limited (Level C), with most support from traditional use and preliminary studies.
Mikanolide and dihydromikanolide inhibit bacterial cell wall synthesis and disrupt membrane integrity, contributing to antimicrobial effects. Flavonoids and sesquiterpene lactones modulate inflammatory pathways by inhibiting NF-κB and COX-2, reducing prostaglandin and cytokine production. Wound healing is promoted through enhanced fibroblast proliferation and collagen deposition, likely via TGF-β signaling. The plant's antiplasmodial activity may involve inhibition of heme polymerization in Plasmodium species.
Invasive tropical Asteraceae used widely in traditional medicine across Asia and Pacific for wound healing and fever.
Mikania micrantha, an invasive tropical Asteraceae, is traditionally used across Asia and the Pacific for wound healing, fever, and malaria management. Its primary active compounds include sesquiterpene lactones (mikanolide, dihydromikanolide) and flavonoids, which contribute to antimicrobial, anti-inflammatory, and wound-healing properties. Evidence is limited (Level C), with most support from traditional use and preliminary studies.
Mikanolide and dihydromikanolide inhibit bacterial cell wall synthesis and disrupt membrane integrity, contributing to antimicrobial effects. Flavonoids and sesquiterpene lactones modulate inflammatory pathways by inhibiting NF-κB and COX-2, reducing prostaglandin and cytokine production. Wound healing is promoted through enhanced fibroblast proliferation and collagen deposition, likely via TGF-β signaling. The plant's antiplasmodial activity may involve inhibition of heme polymerization in Plasmodium species.