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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Morinda morindoides
West African climbing plant used in traditional medicine across Nigeria and Ghana for malaria, fever, and general infections.
Morinda morindoides is a West African climbing plant traditionally used for malaria, fever, and infections. Modern research indicates antiplasmodial, antimicrobial, anti-inflammatory, and analgesic activities. Key active compounds include morindin, anthraquinones, flavonoids, and alkaloids.
The antiplasmodial activity is attributed to anthraquinones and flavonoids that inhibit heme polymerization and disrupt parasite mitochondrial function. Antimicrobial effects involve membrane disruption and inhibition of bacterial DNA gyrase. Anti-inflammatory and analgesic actions are mediated through inhibition of cyclooxygenase-2 (COX-2) and lipoxygenase (LOX) pathways, reducing prostaglandin and leukotriene synthesis.
West African climbing plant used in traditional medicine across Nigeria and Ghana for malaria, fever, and general infections.
Morinda morindoides is a West African climbing plant traditionally used for malaria, fever, and infections. Modern research indicates antiplasmodial, antimicrobial, anti-inflammatory, and analgesic activities. Key active compounds include morindin, anthraquinones, flavonoids, and alkaloids.
The antiplasmodial activity is attributed to anthraquinones and flavonoids that inhibit heme polymerization and disrupt parasite mitochondrial function. Antimicrobial effects involve membrane disruption and inhibition of bacterial DNA gyrase. Anti-inflammatory and analgesic actions are mediated through inhibition of cyclooxygenase-2 (COX-2) and lipoxygenase (LOX) pathways, reducing prostaglandin and leukotriene synthesis.