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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Commiphora myrrha
Ancient resin used since Egyptian times for wound healing, oral care, and religious ceremonies. One of the gifts brought to the infant Jesus.
Myrrh (Commiphora myrrha) is an ancient resin revered for its wound-healing, oral care, and ceremonial uses, with modern evidence supporting its efficacy in gingivitis and inflammation. Its primary active compounds include furanosesquiterpenes such as curzerene and lindestrene, which contribute to its antimicrobial and anti-inflammatory properties.
Myrrh's furanosesquiterpenes, particularly curzerene and lindestrene, inhibit cyclooxygenase-2 (COX-2) and nuclear factor-kappa B (NF-κB) pathways, reducing prostaglandin and pro-inflammatory cytokine production. These compounds also disrupt bacterial cell membranes and quorum sensing, exerting antimicrobial effects against oral pathogens. Additionally, myrrh promotes wound healing by stimulating fibroblast proliferation and collagen synthesis, while its volatile oils provide mild analgesic activity via modulation of transient receptor potential (TRP) channels.
Ancient resin used since Egyptian times for wound healing, oral care, and religious ceremonies. One of the gifts brought to the infant Jesus.
Myrrh (Commiphora myrrha) is an ancient resin revered for its wound-healing, oral care, and ceremonial uses, with modern evidence supporting its efficacy in gingivitis and inflammation. Its primary active compounds include furanosesquiterpenes such as curzerene and lindestrene, which contribute to its antimicrobial and anti-inflammatory properties.
Myrrh's furanosesquiterpenes, particularly curzerene and lindestrene, inhibit cyclooxygenase-2 (COX-2) and nuclear factor-kappa B (NF-κB) pathways, reducing prostaglandin and pro-inflammatory cytokine production. These compounds also disrupt bacterial cell membranes and quorum sensing, exerting antimicrobial effects against oral pathogens. Additionally, myrrh promotes wound healing by stimulating fibroblast proliferation and collagen synthesis, while its volatile oils provide mild analgesic activity via modulation of transient receptor potential (TRP) channels.