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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Panax notoginseng
The Chinese hemostatic herb par excellence; used for both stopping bleeding AND moving blood stasis; critical in trauma, surgery, and cardiovascular formulas.
Panax notoginseng is a premier hemostatic herb in Traditional Chinese Medicine, uniquely capable of both stopping bleeding and resolving blood stasis. Its primary active compounds, notoginsenosides (e.g., R1, Rb1, Rg1) and ginsenosides, along with flavonoids and polysaccharides, contribute to its dual actions in trauma, surgery, and cardiovascular protection.
Notoginsenosides, particularly R1, enhance platelet aggregation and clot formation via activation of thrombin receptors and upregulation of von Willebrand factor, while simultaneously preventing excessive thrombosis through inhibition of platelet P2Y12 receptors and suppression of thromboxane A2 synthesis. The herb also exerts anti-inflammatory effects by downregulating NF-κB and MAPK pathways, reducing TNF-α and IL-6. Additionally, it promotes vasodilation via nitric oxide production and protects against ischemia-reperfusion injury through antioxidant and anti-apoptotic mechanisms.
The Chinese hemostatic herb par excellence; used for both stopping bleeding AND moving blood stasis; critical in trauma, surgery, and cardiovascular formulas.
Panax notoginseng is a premier hemostatic herb in Traditional Chinese Medicine, uniquely capable of both stopping bleeding and resolving blood stasis. Its primary active compounds, notoginsenosides (e.g., R1, Rb1, Rg1) and ginsenosides, along with flavonoids and polysaccharides, contribute to its dual actions in trauma, surgery, and cardiovascular protection.
Notoginsenosides, particularly R1, enhance platelet aggregation and clot formation via activation of thrombin receptors and upregulation of von Willebrand factor, while simultaneously preventing excessive thrombosis through inhibition of platelet P2Y12 receptors and suppression of thromboxane A2 synthesis. The herb also exerts anti-inflammatory effects by downregulating NF-κB and MAPK pathways, reducing TNF-α and IL-6. Additionally, it promotes vasodilation via nitric oxide production and protects against ischemia-reperfusion injury through antioxidant and anti-apoptotic mechanisms.