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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Passiflora caerulea
Hardy ornamental passion flower from South America widely cultivated in temperate gardens, sedative properties.
Passiflora caerulea (blue passionflower) is a hardy ornamental vine native to South America, widely cultivated in temperate gardens for its sedative and anxiolytic properties. Its primary active compounds include flavonoids such as chrysin and vitexin, along with beta-carboline alkaloids and maltol, which collectively contribute to its calming effects. Used traditionally as a nervine for anxiety, insomnia, and muscle tension, its clinical evidence is limited to Level C (pilot studies and small trials).
The sedative and anxiolytic actions of Passiflora caerulea are mediated through multiple pathways. Chrysin and vitexin act as partial agonists at the benzodiazepine binding site of GABA-A receptors, enhancing chloride ion conductance and neuronal inhibition. Beta-carboline alkaloids (e.g., harman) inhibit monoamine oxidase (MAO), increasing synaptic levels of serotonin and other monoamines. Maltol exhibits mild sedative effects via modulation of calcium channels. These combined mechanisms produce a calming effect without significant respiratory depression or cognitive impairment.
Hardy ornamental passion flower from South America widely cultivated in temperate gardens, sedative properties.
Passiflora caerulea (blue passionflower) is a hardy ornamental vine native to South America, widely cultivated in temperate gardens for its sedative and anxiolytic properties. Its primary active compounds include flavonoids such as chrysin and vitexin, along with beta-carboline alkaloids and maltol, which collectively contribute to its calming effects. Used traditionally as a nervine for anxiety, insomnia, and muscle tension, its clinical evidence is limited to Level C (pilot studies and small trials).
The sedative and anxiolytic actions of Passiflora caerulea are mediated through multiple pathways. Chrysin and vitexin act as partial agonists at the benzodiazepine binding site of GABA-A receptors, enhancing chloride ion conductance and neuronal inhibition. Beta-carboline alkaloids (e.g., harman) inhibit monoamine oxidase (MAO), increasing synaptic levels of serotonin and other monoamines. Maltol exhibits mild sedative effects via modulation of calcium channels. These combined mechanisms produce a calming effect without significant respiratory depression or cognitive impairment.