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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Palmitoylethanolamide natural
Endogenous fatty acid amide with analgesic and neuroprotective action.
Palmitoylethanolamide (PEA) is an endogenous fatty acid amide that acts as an endocannabinoid-like compound, primarily used for chronic pain, neuropathy, and inflammation. It exhibits analgesic and neuroprotective properties through modulation of the endocannabinoid system and mast cell stabilization. Key active compounds include palmitoylethanolamide itself, which binds to PPAR-α and other receptors.
PEA exerts its effects by activating peroxisome proliferator-activated receptor alpha (PPAR-α), which downregulates pro-inflammatory cytokines and reduces glial activation. It also enhances endocannabinoid tone by inhibiting fatty acid amide hydrolase (FAAH), increasing anandamide levels. Additionally, PEA stabilizes mast cells by reducing degranulation and release of inflammatory mediators, contributing to its analgesic and neuroprotective actions.
Endogenous fatty acid amide with analgesic and neuroprotective action.
Palmitoylethanolamide (PEA) is an endogenous fatty acid amide that acts as an endocannabinoid-like compound, primarily used for chronic pain, neuropathy, and inflammation. It exhibits analgesic and neuroprotective properties through modulation of the endocannabinoid system and mast cell stabilization. Key active compounds include palmitoylethanolamide itself, which binds to PPAR-α and other receptors.
PEA exerts its effects by activating peroxisome proliferator-activated receptor alpha (PPAR-α), which downregulates pro-inflammatory cytokines and reduces glial activation. It also enhances endocannabinoid tone by inhibiting fatty acid amide hydrolase (FAAH), increasing anandamide levels. Additionally, PEA stabilizes mast cells by reducing degranulation and release of inflammatory mediators, contributing to its analgesic and neuroprotective actions.