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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Physalis alkekengi
Ornamental plant with papery orange husks used in European and Chinese folk medicine for urinary conditions and fever.
Physalis alkekengi, commonly known as Chinese lantern, is used in European and Chinese folk medicine primarily as a diuretic, anti-inflammatory, and antidiabetic agent. Its key active compounds include physalin B, withaphysalin, and other withanolides, which contribute to its pharmacological effects. Evidence for its efficacy is limited (Level C), with most data derived from pilot studies and traditional use.
Physalin B and withanolides inhibit NF-κB and COX-2 pathways, reducing pro-inflammatory cytokines (e.g., TNF-α, IL-6) and prostaglandin synthesis, thereby exerting anti-inflammatory effects. The diuretic action is attributed to citric acid and other organic acids that increase renal sodium and water excretion. Antidiabetic effects may involve activation of AMPK and PPARγ, improving insulin sensitivity and glucose uptake, as shown in preliminary in vitro and animal studies.
Ornamental plant with papery orange husks used in European and Chinese folk medicine for urinary conditions and fever.
Physalis alkekengi, commonly known as Chinese lantern, is used in European and Chinese folk medicine primarily as a diuretic, anti-inflammatory, and antidiabetic agent. Its key active compounds include physalin B, withaphysalin, and other withanolides, which contribute to its pharmacological effects. Evidence for its efficacy is limited (Level C), with most data derived from pilot studies and traditional use.
Physalin B and withanolides inhibit NF-κB and COX-2 pathways, reducing pro-inflammatory cytokines (e.g., TNF-α, IL-6) and prostaglandin synthesis, thereby exerting anti-inflammatory effects. The diuretic action is attributed to citric acid and other organic acids that increase renal sodium and water excretion. Antidiabetic effects may involve activation of AMPK and PPARγ, improving insulin sensitivity and glucose uptake, as shown in preliminary in vitro and animal studies.