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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Piscidia erythrina
Alternative dogwood species with similar analgesic effects.
Piscidia erythrina, also known as Jamaican dogwood, is a medicinal herb traditionally used for neuralgia, sciatica, and insomnia. Modern evidence supports its use as a nerve pain reliever, muscle relaxant, and sedative, attributed to active compounds such as rotenoids, isoflavones, and piscidic acid. Its analgesic and sedative effects are primarily mediated through GABAergic modulation and anti-inflammatory pathways.
The analgesic and sedative effects of Piscidia erythrina are primarily attributed to rotenoids and isoflavones, which potentiate GABA-A receptor activity, leading to central nervous system depression and muscle relaxation. Additionally, piscidic acid inhibits cyclooxygenase (COX) enzymes, reducing prostaglandin synthesis and inflammation. These compounds also modulate voltage-gated sodium channels, contributing to nerve pain relief. The overall mechanism involves synergistic actions on GABAergic, opioid, and serotonergic pathways.
Alternative dogwood species with similar analgesic effects.
Piscidia erythrina, also known as Jamaican dogwood, is a medicinal herb traditionally used for neuralgia, sciatica, and insomnia. Modern evidence supports its use as a nerve pain reliever, muscle relaxant, and sedative, attributed to active compounds such as rotenoids, isoflavones, and piscidic acid. Its analgesic and sedative effects are primarily mediated through GABAergic modulation and anti-inflammatory pathways.
The analgesic and sedative effects of Piscidia erythrina are primarily attributed to rotenoids and isoflavones, which potentiate GABA-A receptor activity, leading to central nervous system depression and muscle relaxation. Additionally, piscidic acid inhibits cyclooxygenase (COX) enzymes, reducing prostaglandin synthesis and inflammation. These compounds also modulate voltage-gated sodium channels, contributing to nerve pain relief. The overall mechanism involves synergistic actions on GABAergic, opioid, and serotonergic pathways.