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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Prunus persica
Peach tree whose leaves, bark, and flowers are used in folk medicine for digestive, respiratory, and kidney conditions.
Prunus persica (peach tree) leaves, bark, and flowers are used in folk medicine primarily as a digestive sedative, antitussive, and diuretic. Its key active compounds include the cyanogenic glycoside amygdalin, along with flavonoids and tannins, which contribute to its traditional applications for respiratory and kidney conditions. Evidence for these uses is limited (Level C), and caution is warranted due to cyanide precursors in seeds and potential toxicity.
Amygdalin, a cyanogenic glycoside, is hydrolyzed to hydrogen cyanide, which may suppress cough reflexes via central nervous system action, though this mechanism is dose-dependent and carries toxicity risks. Flavonoids and tannins provide anti-inflammatory and astringent effects, potentially soothing gastrointestinal mucosa and reducing smooth muscle spasms, supporting its traditional use as a digestive sedative. Diuretic effects are likely mediated by flavonoid-induced renal vasodilation and increased urine output, but clinical data are sparse.
Peach tree whose leaves, bark, and flowers are used in folk medicine for digestive, respiratory, and kidney conditions.
Prunus persica (peach tree) leaves, bark, and flowers are used in folk medicine primarily as a digestive sedative, antitussive, and diuretic. Its key active compounds include the cyanogenic glycoside amygdalin, along with flavonoids and tannins, which contribute to its traditional applications for respiratory and kidney conditions. Evidence for these uses is limited (Level C), and caution is warranted due to cyanide precursors in seeds and potential toxicity.
Amygdalin, a cyanogenic glycoside, is hydrolyzed to hydrogen cyanide, which may suppress cough reflexes via central nervous system action, though this mechanism is dose-dependent and carries toxicity risks. Flavonoids and tannins provide anti-inflammatory and astringent effects, potentially soothing gastrointestinal mucosa and reducing smooth muscle spasms, supporting its traditional use as a digestive sedative. Diuretic effects are likely mediated by flavonoid-induced renal vasodilation and increased urine output, but clinical data are sparse.