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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Pteridium aquilinum
Globally distributed fern; young fronds eaten in Japan and Korea; rhizome used for starch; carcinogenic with long-term use.
Pteridium aquilinum (bracken fern) is a globally distributed plant whose young fronds are traditionally consumed as food in East Asia, while its rhizome provides starch. Its primary active compound, ptaquiloside, is a potent carcinogen, and the plant also contains thiaminase, which can cause thiamine deficiency. Due to these risks, it is not recommended for medicinal or regular dietary use.
Ptaquiloside, a norsesquiterpene glycoside, undergoes metabolic activation to form a DNA-alkylating diol epoxide that induces mutations in the TP53 gene, contributing to carcinogenesis. Thiaminase degrades thiamine (vitamin B1), leading to beriberi-like neurological symptoms with chronic ingestion. Tannins may provide mild astringent effects, but no therapeutic receptor-mediated pathways are established for clinical use.
Globally distributed fern; young fronds eaten in Japan and Korea; rhizome used for starch; carcinogenic with long-term use.
Pteridium aquilinum (bracken fern) is a globally distributed plant whose young fronds are traditionally consumed as food in East Asia, while its rhizome provides starch. Its primary active compound, ptaquiloside, is a potent carcinogen, and the plant also contains thiaminase, which can cause thiamine deficiency. Due to these risks, it is not recommended for medicinal or regular dietary use.
Ptaquiloside, a norsesquiterpene glycoside, undergoes metabolic activation to form a DNA-alkylating diol epoxide that induces mutations in the TP53 gene, contributing to carcinogenesis. Thiaminase degrades thiamine (vitamin B1), leading to beriberi-like neurological symptoms with chronic ingestion. Tannins may provide mild astringent effects, but no therapeutic receptor-mediated pathways are established for clinical use.