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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Quercus infectoria
Oak producing tannin-rich galls used across the Middle East and Unani medicine for diarrhea, skin conditions, and antimicrobial.
Quercus infectoria (Aleppo oak) galls are a traditional astringent remedy in Unani and Middle Eastern medicine, valued for their high tannin content (tannic, gallic, ellagic acids) and used topically or internally for diarrhea, wound healing, and antimicrobial effects. Evidence is limited to pilot studies and traditional use (Level C).
The primary mechanism is astringent action via tannins, which precipitate proteins in mucosal membranes, forming a protective layer that reduces secretion and inflammation. Gallic acid and ellagic acid inhibit cyclooxygenase (COX) and lipoxygenase (LOX) pathways, decreasing prostaglandin and leukotriene synthesis. Tannic acid also chelates metal ions, disrupting bacterial cell walls and inhibiting microbial growth. Beta-sitosterol contributes mild anti-inflammatory activity by modulating cytokine release.
Oak producing tannin-rich galls used across the Middle East and Unani medicine for diarrhea, skin conditions, and antimicrobial.
Quercus infectoria (Aleppo oak) galls are a traditional astringent remedy in Unani and Middle Eastern medicine, valued for their high tannin content (tannic, gallic, ellagic acids) and used topically or internally for diarrhea, wound healing, and antimicrobial effects. Evidence is limited to pilot studies and traditional use (Level C).
The primary mechanism is astringent action via tannins, which precipitate proteins in mucosal membranes, forming a protective layer that reduces secretion and inflammation. Gallic acid and ellagic acid inhibit cyclooxygenase (COX) and lipoxygenase (LOX) pathways, decreasing prostaglandin and leukotriene synthesis. Tannic acid also chelates metal ions, disrupting bacterial cell walls and inhibiting microbial growth. Beta-sitosterol contributes mild anti-inflammatory activity by modulating cytokine release.