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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Ribes nigrum
European fruit with exceptionally high vitamin C; leaves and berries used medicinally for inflammation.
Ribes nigrum (black currant) is a European fruit whose leaves and berries are used medicinally for their exceptionally high vitamin C content and anti-inflammatory properties. Its primary uses include supporting joint health, immune function, and reducing oxidative stress, with key active compounds including anthocyanins, gamma-linolenic acid, quercetin, and myricetin.
The anti-inflammatory effects of Ribes nigrum are mediated through inhibition of cyclooxygenase-2 (COX-2) and lipoxygenase pathways by anthocyanins and flavonols, reducing prostaglandin and leukotriene synthesis. Gamma-linolenic acid (GLA) serves as a precursor to anti-inflammatory prostaglandin E1, while vitamin C and other antioxidants scavenge reactive oxygen species and modulate nuclear factor-kappa B (NF-κB) signaling, thereby attenuating inflammatory cytokine production.
European fruit with exceptionally high vitamin C; leaves and berries used medicinally for inflammation.
Ribes nigrum (black currant) is a European fruit whose leaves and berries are used medicinally for their exceptionally high vitamin C content and anti-inflammatory properties. Its primary uses include supporting joint health, immune function, and reducing oxidative stress, with key active compounds including anthocyanins, gamma-linolenic acid, quercetin, and myricetin.
The anti-inflammatory effects of Ribes nigrum are mediated through inhibition of cyclooxygenase-2 (COX-2) and lipoxygenase pathways by anthocyanins and flavonols, reducing prostaglandin and leukotriene synthesis. Gamma-linolenic acid (GLA) serves as a precursor to anti-inflammatory prostaglandin E1, while vitamin C and other antioxidants scavenge reactive oxygen species and modulate nuclear factor-kappa B (NF-κB) signaling, thereby attenuating inflammatory cytokine production.