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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Syzygium polyanthum
Indonesian aromatic leaf used as a spice and medicine in Jamu for diabetes management, hypertension, and digestive conditions.
Salam Leaf (Syzygium polyanthum) is an aromatic leaf used in Indonesian Jamu for diabetes, hypertension, and digestive health. Its primary active compounds include quercetin, myricetin, kaempferol, and ellagic acid, which contribute to its antidiabetic, antihypertensive, and anti-inflammatory effects. Evidence level C indicates limited clinical studies but strong traditional use.
The antidiabetic effects are mediated through inhibition of α-glucosidase and α-amylase enzymes, reducing postprandial glucose absorption, and via activation of AMPK and PPARγ pathways to improve insulin sensitivity. The antihypertensive action involves vasodilation through nitric oxide (NO) production and calcium channel blockade, attributed to flavonoids like quercetin and myricetin. Anti-inflammatory properties result from inhibition of NF-κB and COX-2, reducing pro-inflammatory cytokines.
Indonesian aromatic leaf used as a spice and medicine in Jamu for diabetes management, hypertension, and digestive conditions.
Salam Leaf (Syzygium polyanthum) is an aromatic leaf used in Indonesian Jamu for diabetes, hypertension, and digestive health. Its primary active compounds include quercetin, myricetin, kaempferol, and ellagic acid, which contribute to its antidiabetic, antihypertensive, and anti-inflammatory effects. Evidence level C indicates limited clinical studies but strong traditional use.
The antidiabetic effects are mediated through inhibition of α-glucosidase and α-amylase enzymes, reducing postprandial glucose absorption, and via activation of AMPK and PPARγ pathways to improve insulin sensitivity. The antihypertensive action involves vasodilation through nitric oxide (NO) production and calcium channel blockade, attributed to flavonoids like quercetin and myricetin. Anti-inflammatory properties result from inhibition of NF-κB and COX-2, reducing pro-inflammatory cytokines.