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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Salix matsudana
A Chinese ornamental willow with salicin-containing bark used in TCM for pain, fever, and rheumatic conditions.
Salix matsudana, a Chinese ornamental willow, is traditionally used in TCM for pain, fever, and rheumatism due to its salicin-rich bark. Its primary active compounds include salicin, flavonoids, and tannins, which contribute to anti-inflammatory and analgesic effects. Evidence is limited (Level C), supporting its use for mild pain and inflammation.
Salicin, a prodrug, is metabolized to salicylic acid, which irreversibly inhibits cyclooxygenase (COX-1 and COX-2) enzymes, reducing prostaglandin synthesis and thereby alleviating pain and fever. Flavonoids provide additional anti-inflammatory activity by inhibiting NF-κB activation and modulating cytokine release (e.g., TNF-α, IL-6). Tannins may contribute astringent effects but have unclear systemic anti-inflammatory roles.
A Chinese ornamental willow with salicin-containing bark used in TCM for pain, fever, and rheumatic conditions.
Salix matsudana, a Chinese ornamental willow, is traditionally used in TCM for pain, fever, and rheumatism due to its salicin-rich bark. Its primary active compounds include salicin, flavonoids, and tannins, which contribute to anti-inflammatory and analgesic effects. Evidence is limited (Level C), supporting its use for mild pain and inflammation.
Salicin, a prodrug, is metabolized to salicylic acid, which irreversibly inhibits cyclooxygenase (COX-1 and COX-2) enzymes, reducing prostaglandin synthesis and thereby alleviating pain and fever. Flavonoids provide additional anti-inflammatory activity by inhibiting NF-κB activation and modulating cytokine release (e.g., TNF-α, IL-6). Tannins may contribute astringent effects but have unclear systemic anti-inflammatory roles.