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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Salix purpurea
Purple-barked willow species with high salicin content used in European folk medicine for pain, fever, and inflammation.
Salix purpurea (purple willow) is a botanical source of salicin and related salicylates, traditionally used in European folk medicine for pain, fever, and inflammation. Its primary active compounds include salicin, fragilin, populin, and tannins, which collectively provide analgesic, anti-inflammatory, and antipyretic effects. Modern evidence supports its use for mild to moderate pain and inflammatory conditions, though clinical data remain limited.
Salicin, the primary active glycoside, is hydrolyzed in the gastrointestinal tract to saligenin, which is then oxidized to salicylic acid in the liver and blood. Salicylic acid inhibits cyclooxygenase (COX-1 and COX-2) enzymes, reducing prostaglandin synthesis and thereby alleviating pain, fever, and inflammation. Additional phenolic glycosides (fragilin, populin) may contribute to anti-inflammatory activity via modulation of nuclear factor-kappa B (NF-κB) and inhibition of leukotriene formation. Tannins provide astringent and mild antioxidant effects.
Purple-barked willow species with high salicin content used in European folk medicine for pain, fever, and inflammation.
Salix purpurea (purple willow) is a botanical source of salicin and related salicylates, traditionally used in European folk medicine for pain, fever, and inflammation. Its primary active compounds include salicin, fragilin, populin, and tannins, which collectively provide analgesic, anti-inflammatory, and antipyretic effects. Modern evidence supports its use for mild to moderate pain and inflammatory conditions, though clinical data remain limited.
Salicin, the primary active glycoside, is hydrolyzed in the gastrointestinal tract to saligenin, which is then oxidized to salicylic acid in the liver and blood. Salicylic acid inhibits cyclooxygenase (COX-1 and COX-2) enzymes, reducing prostaglandin synthesis and thereby alleviating pain, fever, and inflammation. Additional phenolic glycosides (fragilin, populin) may contribute to anti-inflammatory activity via modulation of nuclear factor-kappa B (NF-κB) and inhibition of leukotriene formation. Tannins provide astringent and mild antioxidant effects.