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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Flueggea virosa
Small African and Asian shrub used across tropical Africa in traditional medicine for malaria, skin conditions, and as an emetic.
Securinega virosa (Flueggea virosa) is a shrub used in African traditional medicine for malaria, skin conditions, and as an emetic. Modern research supports its antiplasmodial, antimicrobial, and wound-healing properties, attributed to alkaloids such as securinine, phyllanthidine, and norsecurinine, along with flavonoids. Its clinical evidence remains limited (Grade C), with most data derived from in vitro and preclinical studies.
Securinine acts as a competitive antagonist at GABA-A receptors, leading to central nervous system excitation and convulsant effects at high doses. Antiplasmodial activity is mediated through inhibition of heme polymerization and disruption of parasite mitochondrial function. Antimicrobial effects involve membrane disruption and inhibition of bacterial efflux pumps. Wound healing is associated with enhanced collagen synthesis and fibroblast proliferation, likely via flavonoid-induced TGF-β signaling.
Small African and Asian shrub used across tropical Africa in traditional medicine for malaria, skin conditions, and as an emetic.
Securinega virosa (Flueggea virosa) is a shrub used in African traditional medicine for malaria, skin conditions, and as an emetic. Modern research supports its antiplasmodial, antimicrobial, and wound-healing properties, attributed to alkaloids such as securinine, phyllanthidine, and norsecurinine, along with flavonoids. Its clinical evidence remains limited (Grade C), with most data derived from in vitro and preclinical studies.
Securinine acts as a competitive antagonist at GABA-A receptors, leading to central nervous system excitation and convulsant effects at high doses. Antiplasmodial activity is mediated through inhibition of heme polymerization and disruption of parasite mitochondrial function. Antimicrobial effects involve membrane disruption and inhibition of bacterial efflux pumps. Wound healing is associated with enhanced collagen synthesis and fibroblast proliferation, likely via flavonoid-induced TGF-β signaling.