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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Silphium perfoliatum
Large North American prairie herb used by Native Americans as medicine for fever, rheumatism, and liver support.
Silphium perfoliatum (cup plant) is a North American perennial traditionally used by Native Americans for fever, rheumatism, and liver complaints. Modern research suggests anti-inflammatory, digestive, and antipyretic properties, attributed to sesquiterpene lactones, flavonoids, and silphiperfolene. Evidence is limited (Grade C) to traditional use.
Sesquiterpene lactones (e.g., silphiperfolene) inhibit NF-κB and COX-2 pathways, reducing pro-inflammatory cytokines (TNF-α, IL-6). Flavonoids contribute antioxidant activity and may modulate CYP450 enzymes. The bitter principles stimulate digestive secretions via vagal activation. Antipyretic effects are likely mediated through prostaglandin synthesis inhibition in the hypothalamus.
Large North American prairie herb used by Native Americans as medicine for fever, rheumatism, and liver support.
Silphium perfoliatum (cup plant) is a North American perennial traditionally used by Native Americans for fever, rheumatism, and liver complaints. Modern research suggests anti-inflammatory, digestive, and antipyretic properties, attributed to sesquiterpene lactones, flavonoids, and silphiperfolene. Evidence is limited (Grade C) to traditional use.
Sesquiterpene lactones (e.g., silphiperfolene) inhibit NF-κB and COX-2 pathways, reducing pro-inflammatory cytokines (TNF-α, IL-6). Flavonoids contribute antioxidant activity and may modulate CYP450 enzymes. The bitter principles stimulate digestive secretions via vagal activation. Antipyretic effects are likely mediated through prostaglandin synthesis inhibition in the hypothalamus.