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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Solanum indicum
Ayurvedic herb in Dashamoola group; spiny shrub for respiratory and rheumatic conditions.
Solanum indicum, a spiny shrub from the Solanaceae family, is a key component of the Ayurvedic Dashamoola group. It is traditionally used for respiratory and rheumatic conditions, with modern applications as a bronchodilator, anti-inflammatory, antipyretic, and antimicrobial agent. Its primary active compounds include solasodin, diosgenin, solanine, alkaloids, sterols, and saponins.
The bronchodilator effect is mediated by solasodin and diosgenin, which may act as beta-2 adrenergic receptor agonists and phosphodiesterase inhibitors, leading to bronchial smooth muscle relaxation. Anti-inflammatory actions involve inhibition of cyclooxygenase (COX) and lipoxygenase (LOX) pathways, reducing prostaglandin and leukotriene synthesis. The antipyretic effect is likely due to central prostaglandin inhibition, while antimicrobial activity results from alkaloids and saponins disrupting microbial cell membranes.
Ayurvedic herb in Dashamoola group; spiny shrub for respiratory and rheumatic conditions.
Solanum indicum, a spiny shrub from the Solanaceae family, is a key component of the Ayurvedic Dashamoola group. It is traditionally used for respiratory and rheumatic conditions, with modern applications as a bronchodilator, anti-inflammatory, antipyretic, and antimicrobial agent. Its primary active compounds include solasodin, diosgenin, solanine, alkaloids, sterols, and saponins.
The bronchodilator effect is mediated by solasodin and diosgenin, which may act as beta-2 adrenergic receptor agonists and phosphodiesterase inhibitors, leading to bronchial smooth muscle relaxation. Anti-inflammatory actions involve inhibition of cyclooxygenase (COX) and lipoxygenase (LOX) pathways, reducing prostaglandin and leukotriene synthesis. The antipyretic effect is likely due to central prostaglandin inhibition, while antimicrobial activity results from alkaloids and saponins disrupting microbial cell membranes.