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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Aralia racemosa
Eastern North American herb used by Iroquois, Cherokee, and Menominee as a respiratory tonic, adaptogen, and treatment for back pain and rheumatism.
Spikenard Native (Aralia racemosa) is a traditional North American herb used by Indigenous peoples as a respiratory tonic, adaptogen, and for musculoskeletal pain. Its primary active compounds include araloside A, oleanolic acid, and kaurenoic acid, which contribute to its expectorant, anti-inflammatory, and adaptogenic properties.
Aralia racemosa exhibits anti-inflammatory activity through inhibition of cyclooxygenase (COX) and lipoxygenase (LOX) pathways, reducing prostaglandin and leukotriene synthesis. Its triterpenoid saponins, particularly araloside A, modulate the hypothalamic-pituitary-adrenal (HPA) axis, supporting adaptogenic effects. Expectorant action is attributed to saponin-induced irritation of gastric mucosa, reflexively increasing respiratory tract secretions. Kaurenoic acid demonstrates analgesic effects via interaction with TRPV1 receptors and modulation of pain signaling.
Eastern North American herb used by Iroquois, Cherokee, and Menominee as a respiratory tonic, adaptogen, and treatment for back pain and rheumatism.
Spikenard Native (Aralia racemosa) is a traditional North American herb used by Indigenous peoples as a respiratory tonic, adaptogen, and for musculoskeletal pain. Its primary active compounds include araloside A, oleanolic acid, and kaurenoic acid, which contribute to its expectorant, anti-inflammatory, and adaptogenic properties.
Aralia racemosa exhibits anti-inflammatory activity through inhibition of cyclooxygenase (COX) and lipoxygenase (LOX) pathways, reducing prostaglandin and leukotriene synthesis. Its triterpenoid saponins, particularly araloside A, modulate the hypothalamic-pituitary-adrenal (HPA) axis, supporting adaptogenic effects. Expectorant action is attributed to saponin-induced irritation of gastric mucosa, reflexively increasing respiratory tract secretions. Kaurenoic acid demonstrates analgesic effects via interaction with TRPV1 receptors and modulation of pain signaling.