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Sonchus arvensis
Southeast Asian herb used in Jamu for kidney stones, urinary conditions, and hypertension; one of the most popular Indonesian herbal kidney remedies.
Tempuyung (Sonchus arvensis) is a Southeast Asian herb widely used in Jamu for kidney stones, urinary conditions, and hypertension. Its primary active compounds include flavonoids such as luteolin-7-glucoside, apigenin-7-glucoside, and quercetin, along with inulin. Modern evidence supports its antilithic, diuretic, and antihypertensive properties, though clinical data remain limited.
Tempuyung exhibits diuretic effects by increasing renal excretion of sodium and water, likely through inhibition of the Na⁺-K⁺-2Cl⁻ cotransporter in the loop of Henle. Its antilithic action involves inhibition of calcium oxalate crystal aggregation and growth, mediated by flavonoids like luteolin-7-glucoside. Antihypertensive effects may be due to ACE inhibition and vasodilation via nitric oxide pathways. Anti-inflammatory activity is attributed to suppression of COX-2 and LOX enzymes by quercetin and apigenin derivatives.
Southeast Asian herb used in Jamu for kidney stones, urinary conditions, and hypertension; one of the most popular Indonesian herbal kidney remedies.
Tempuyung (Sonchus arvensis) is a Southeast Asian herb widely used in Jamu for kidney stones, urinary conditions, and hypertension. Its primary active compounds include flavonoids such as luteolin-7-glucoside, apigenin-7-glucoside, and quercetin, along with inulin. Modern evidence supports its antilithic, diuretic, and antihypertensive properties, though clinical data remain limited.
Tempuyung exhibits diuretic effects by increasing renal excretion of sodium and water, likely through inhibition of the Na⁺-K⁺-2Cl⁻ cotransporter in the loop of Henle. Its antilithic action involves inhibition of calcium oxalate crystal aggregation and growth, mediated by flavonoids like luteolin-7-glucoside. Antihypertensive effects may be due to ACE inhibition and vasodilation via nitric oxide pathways. Anti-inflammatory activity is attributed to suppression of COX-2 and LOX enzymes by quercetin and apigenin derivatives.