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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Anticlea elegans
Toxic white-flowered bulb of North American meadows often confused with edible camas, causes livestock poisoning.
Zigadenus elegans, commonly known as death camas, is a highly toxic perennial herb found in North American meadows. It contains steroidal alkaloids such as zygacine and veratrum-type alkaloids that act as sodium channel agonists, leading to severe poisoning. Despite historical confusion with edible camas, it has no medicinal uses and is primarily studied for its toxicological properties.
The primary toxic mechanism involves activation of voltage-gated sodium channels by zygacine and related veratrum alkaloids, causing persistent depolarization of excitable membranes. This leads to excessive salivation, muscle weakness, convulsions, and potentially fatal cardiac or respiratory failure. The alkaloids also interfere with neurotransmission at autonomic ganglia and neuromuscular junctions, amplifying cholinergic and adrenergic effects.
Toxic white-flowered bulb of North American meadows often confused with edible camas, causes livestock poisoning.
Zigadenus elegans, commonly known as death camas, is a highly toxic perennial herb found in North American meadows. It contains steroidal alkaloids such as zygacine and veratrum-type alkaloids that act as sodium channel agonists, leading to severe poisoning. Despite historical confusion with edible camas, it has no medicinal uses and is primarily studied for its toxicological properties.
The primary toxic mechanism involves activation of voltage-gated sodium channels by zygacine and related veratrum alkaloids, causing persistent depolarization of excitable membranes. This leads to excessive salivation, muscle weakness, convulsions, and potentially fatal cardiac or respiratory failure. The alkaloids also interfere with neurotransmission at autonomic ganglia and neuromuscular junctions, amplifying cholinergic and adrenergic effects.